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[5] [6] [7] [8] Biomedical research has revealed many similarities between these diseases at the subcellular level, including atypical protein assemblies (like proteinopathy) and induced cell death
In a model of tumor dormancy, leukemia cells exhibited prolonged survival by upregulating the expression of B7-H1 and B7.1, thereby evading CTL-mediated destruction [216]
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