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firstly demonstrates the utility of a novel metabolic agent, GLP-1, in attenuating myocardial stunning after ischemiareperfusion in canine models [97]

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Lau J, Bloch P, Schaffer L, Pettersson I, Spetzler J, Kofoed J, Madsen K, Knudsen LB, McGuire J, Steensgaard DB, Strauss HM, Gram DX, Knudsen SM, Nielsen FS, Thygesen P, Reedtz-Runge S, Kruse T (2015) Discovery of the once-weekly glucagon-like peptide-1 (GLP-1) analogue semaglutide

PRMT1 expression was elevated in fibroblasts from IPF patients and BLM-treated mice, but not in total lung tissue, suggesting cell-type specificity [90]

Studies suggest 10-20 times higher tissue concentrations compared to topical application of equivalent amounts

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