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A bacterial toxin that controls cell cycle progression as a deoxyribonuclease I-like protein

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Interestingly, several in vitro studies of experimentally induced G6PD-d in human monocytes and endothelial cells have demonstrated that exposure to hyperglycemia or free fatty acids alone, as seen in diabetes or hyperlipidemia, decrease G6PD activity

doi: 10.7326/m14-2385 18 RouseBChaimaniALiT

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