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ISBN 978-1770073531
MOTS-c, by contrast, works from the inside of the mitochondria outward, activating the same primordial energy-sensing cascade that millions of years of evolution have refined into the body's most fundamental metabolic regulator
In mice, up-regulation of BAP1 delays AKI and preserves renal tissues[18]
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Overall the above reported evidence on the involvement of GSH and the redox environment are only some issues that deserve to be further investigated to comprehend the several aspects characterizing the possible crosstalk between redox reactions and modulation of ATG
Journal of the European Academy of Dermatology and Venereology: JEADV
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