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A contribution of NTS astrocytes to the appetite suppression caused by acute GLP-1R agonists also seems possible from evidence showing (1) expression of GLP-1Rs in this cell type, (2) functional responses of astrocytes to exendin-4 and liraglutide, and (3) negation of appetite suppression from administration of exendin-4 into the NTS when astrocyte metabolism is selectively inhibited in this region [56]

These results on the thalamic and rTPJ Gln levels remained significant after excluding one individual with comorbid schizophrenia from the ASD group and another with comorbid mild intellectual disability

Showed that the fragment increased fat oxidation in ob/ob mice without changing lean body mass or serum IGF-1 levels [1]

Because of its essential role in cellular wellness, glutathione is often described as one of the bodys most important internal antioxidants

Role of oxidative stress in cardiac dysfunction and subcellular defects due to ischemia-reperfusion injury

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