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Although there are currently no corresponding agonists or inhibitors for these potential targets, functional studies of these potential biomolecules using existing techniques at the gene, protein, and cellular levels are crucial for identifying drug candidates and delaying or reversing disease progression
PMID: 32861979
Chiang, P
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Crucially, HBV independently suppresses DNA repair capacity by downregulating poly(ADP-ribose) polymerase 1 (PARP1) via YTHDF2-mediated m6A methylationwithout enhancing ROS production or altering oxidative stress pathways [389, 391]
M., Quinn J
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