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Mechanistic analysis revealed that these PDEVs penetrate the suprabasal epidermal layers and deliver payloads that suppress IL-17/Th17 inflammatory signaling in keratinocytes by activating the upstream Nrf2 pathway, whose activation induces an antioxidant response and enhances the skin barrier (promoting keratinocyte differentiation and cornification) to counteract inflammatory hyperproliferation in psoriatic skin [227,228,229]
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[Abstract]2024 Aug;43(15):3090-3115
This is the central problem with oral GLP-1s, then
and HGF) [23] or bone marrow stromal cells with the application of cyclosporine, minocycline, and methylprednisolone [24])
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