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Furthermore, since both glucagon and GLP-1 receptors are expressed in the endocrine pancreas, GLP-1R agonists such as semaglutide may also indirectly ameliorate inflammatory injury by regulating various metabolic parameters, including glucose-dependent insulinotropic peptide (GIP), glucagon, insulin, glucose levels, and circulating free fatty acids 16,17

Most of the work has been included in patent no

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